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| *Women health>>>Irritable Bowel Syndrome |
Does Irritable Bowel Syndrome cause your poop to be different shapes? |
Instead of just straight pieces, can they have funny shapes to them? Serious answers only please. Thank you! Absolutely not. One is either constipated or goes often (diarrhoea), but the shape of one's stool depends on it's consistency. No. It is when you can't just drop a normal deuce, you either have diarrhea or constipation. Well, you could sort of say they different shapes unlike normal crap. But I won't go into detail! Irritable bowel is like nervous stomach. It causes you to have diahrea and cramps at the most inopportune times. It's a serious problem and should be taken care of my a medical professional. Hi Suzie, symptoms of Irritable Bowel Syndrome vary quite widely from patient to patient, but there are some common symptoms of Irritable Bowel Syndrome that can indicate an IBS diagnosis. They include: diarrhea, constipation, alternating diarrhea and constipation, stomach pain, bloating, excess gas or wind, nausea. You do not need to have all of these symptoms of Irritable Bowel Syndrome to be diagnosed with the disease, but patients will usually have some diarrhea or constipation plus stomach pain. One of the common symptoms of Irritable Bowel Syndrome in IBS sufferers is that their stomach pain is relieved when they have a bowel movement. They may find that the consistency or shape of their stool changes, and they may also pass some mucus in the stool. It is vital that you are properly diagnosed with IBS by a medical professional, as bowel symptoms of Irritable Bowel Syndrome can be the result of many other health conditions such as celiac disease and inflammatory bowel diseases. It is not possible to accurately self-diagnosis IBS and you may put your health at risk if you do so. I would recommend a thorough examination by your doctor if you have symptoms of Irritable Bowel Syndrome. Jason Homan http://www.askedweb.com/askedweb/categor... The AskedWeb.com information portal ... 禄 I would suggest you be tested for Celiac Disease What is Celiac Disease? Celiac disease (CD) is a genetic disorder. In people with CD, eating certain types of protein, called gluten, sets off an autoimmune response that causes damage to the small intestine. This, in turn, causes the small intestine to lose its ability to absorb the nutrients found in food, leading to malnutrition and a variety of other complications. The offending protein, gluten, is found in wheat, barley, rye, and to a lesser extent, oats (WBRO). Related proteins are found in triticale, spelt, kamut. Refer to grains and flours Glossary for a more extensive list of both safe and offending grains. Celiac Disease is: * a genetic, inheritable disease. * linked to genetically transmitted histocompatibility cell antigens (HLA DR3-DQ2, DR5/7 DQ2, and DR4-DQ8). * COMMON. Approximately 1 in 133 people have CD, however, only about 3% of these have been diagnosed. This means that there are over 2.1 million undiagnosed people with celiac disease in the United States. * characterized by damage to the mucosal lining of the small intestine which is known as villous atrophy. * responsible for the malabsorption of nutrients resulting in malnutrition. * linked to skin blisters known as dermatitis herpetiformis (DH). * not age-dependent. It may become active at any age. Return to top Celiac Disease is NOT: * simply a food allergy. * an idiosyncratic reaction to food proteins (mediated by IgE). * typified by a rapid histamine-type reaction (such as bronchospasm, urticaria, etc.). Return to top The Damaging Proteins The term "gluten" is, in a sense, a generic term for the storage proteins that are found in grains. In reality, each type of protein - gliadin in wheat, secalin in rye, hordein in barley, avenin in oats, zein in corn and oryzenin in rice - is slightly different from the others. The "gluten" in wheat, rye, barley, and in a much lower amount, oats, contains particular amino acid sequences that are harmful to persons with celiac disease. The damaging proteins are particularly rich in proline and glutamine (especially the amino acid sequences which are in the following orders: Pro-Ser-Gln-Gln and Gln-Gln-Gln-Pro). As peptides, some such as 33-MER, cannot be broken down any further. In people with celiac disease, 33-MER stimulates T-cells to produce antibodies. The antibodies, in turn, attack the villi in the small intestine, reducing their ability to absorb nutrients. It is important to note that these sequences are NOT found in the proteins of corn and rice. Return to top The Nature of the Injury The damage to the small intestine (the jejunum) caused by this disease is very slow to develop and is insidious. It is: * almost certainly mediated by the immune system. * associated with ANTIBODIES to gliadin, reticulin and/or endomysial (smooth muscle) proteins. * probably not directly caused by the antibodies, though they may be signals for cell-mediated immunity. * probably produced by the cellular immune system (T cells) - but only when gluten-type prolamins are present. * reversible, in most cases, to completely normal bowel function, if the injurious protein is excluded from the diet. Return to top How Does One "Catch" Celiac Disease? Celiac disease cannot be "caught," but rather the potential for CD may be in the body from birth. Its onset is not confined to a particular age range or gender, although more women are diagnosed than men. It is not known exactly what activates the disease, however three things are required for a person to develop CD: * A genetic disposition: being born with the necessary genes. The Human Leukocyte Antigen (HLA) genes specifically linked to celiac disease are DR3, DQ2 and DQ8. * A trigger: some environmental, emotional or physical event in one鈥檚 life. While triggering factors are not fully understood, possibilities include, but are not limited to adding solids to a baby鈥檚 diet, going through puberty, enduring a surgery or pregnancy, experiencing a stressful situation, catching a virus, increasing WBRO products in the diet, or developing a bacterial infection to which the immune system responds inappropriately. * A diet: containing WBRO, or any of their derivatives. Return to top Summary Celiac disease is life-long and currently incurable. The only known treatment at this time is strict adherence to a gluten-free lifestyle, free of WBRO. How is Celiac Disease Diagnosed? When working with a physician to diagnose and/or confirm celiac disease (CD), three major steps are taken. First, a thorough physical examination is conducted, including a series of blood tests, sometimes referred to as the Celiac Blood Panel. Second, a duodenal biopsy is performed with multiple samples from multiple locations in the small intestine. And third, the gluten-free diet is implemented. When the patient shows a positive response to the diet - symptoms subside and the small intestine returns to its normal, healthy state - the diagnosis of CD is confirmed. (NOTE: To ensure the most accurate and timely diagnosis, the gluten-free diet should be implemented only after the first two steps have been completed.) 1: Examination Patient History When reviewing a patient's medical history and symptoms with a physician, the following areas should be considered in the discussion: (The first three are applicable to adults and children. The last is specific to children.) * What are the symptoms? How long have they been present? How often do they occur? * What is the patient's emotional state? Is it consistent throughout the day? When and for how long do the symptoms occur? * What else is involved? Other diseases? Other organs? * How is the child developing? See What are the Symptoms of Celiac Disease? for a thorough list of possible symptoms. Physical Examination Depending on the presentation of symptoms, the physician will check for some of the following items: * emaciation * pallor (due to anemia) * hypotension (low blood pressure) * edema (due to low levels of protein, [albumin] in the blood) * dermatitis herpetiformis (skin lesions) * easy bruising (lack of vitamin K) * bone or skin and mucosa membrane changes due to vitamin deficiencies * protruding or distended abdomen (intestine dysmotility) * loss of various sensations in extremities including vibration, position and light touch (vitamin deficiency) * signs of severe vitamin/mineral deficiencies which may include: * -diminished deep tendon reflexes * muscle spasms (magnesium and/or calcium deficiency) * bone tenderness and bone pain (due to osteomalacia) Blood Tests A number of tests, sometimes collectively referred to as the Celiac Blood Panel, will aid the physician in diagnosis. The tests may include, but are not limited to: * Serologic Tests 1. EMA (Immunoglobulin A anti-endomysium antibodies) 2. AGA (IgA anti-gliadin antibodies) 3. AGG (IgG anti-gliadin antibodies) 4. tTGA (IgA anti-tissue transglutaminase) * Tolerance or Measure of Digestion/Absorption Tests 1. Lactose tolerance test. 2. D-Xylose test. Return to top 2: Biopsy In the event that clinical signs and laboratory tests indicate probable malabsorption, a biopsy of the small intestine [jejunal] is called for. In this test, a small flexible biopsy instrument is passed through a tube, down the throat, through the stomach and into the upper end of the small intestine where patchy, multiple snippets of tissue are gathered. The tube is removed and the tissue samples are examined under a microscope for signs of injury. chairperson La North Shore Celiac Sprue Association |
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